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Exosome secreted from adipose-derived stem cells attenuates diabetic nephropathy by promoting autophagy flux and inhibiting apoptosis in podocyte.

Mechanism of Regulation of Big-Conductance Ca-Activated K Channels by mTOR Complex 2 in Podocytes.

The natural drug DIAVIT is protective in a type II mouse model of diabetic nephropathy.

Urine Podoplanin Heralds the Onset of Ischemia-Reperfusion Injury of the Kidney.

Prompt apoptotic response to high glucose in SGLT expressing renal cells.

Inhibition of HMGB1 attenuates podocyte apoptosis and epithelial-to-mesenchymal transition (EMT) by regulating autophagy flux.

Podocyturia: why it may have added value in rare diseases.

Diabetes-Induced DUSP4 Reduction Promotes Podocyte Dysfunction and Progression of Diabetic Nephropathy.

Tim-3 aggravates podocyte injury in diabetic nephropathy by promoting macrophage activation via the NF-κB/TNF-α pathway.

Novel tubular-glomerular interplay in diabetic kidney disease mediated by sirtuin 1, nicotinamide mononucleotide, and nicotinamide adenine dinucleotide Oshima Award Address 2017.

[Membranous glomerulonephritis : An example of individualized medicine in nephrology].

Inhibiting 4E-BP1 re-activation represses podocyte cell cycle re-entry and apoptosis induced by adriamycin.

LRG1 Promotes Diabetic Kidney Disease Progression by Enhancing TGF--Induced Angiogenesis.

IQGAP1 mediates podocyte injury in diabetic kidney disease by regulating nephrin endocytosis.

DNMT1 in Progenitor Cells Is Essential for Transposable Element Silencing and Kidney Development.

The beta2-adrenergic receptor (β -AR) in kidney biology: A current prospective.

Single-Cell RNA Profiling of Glomerular Cells Shows Dynamic Changes in Experimental Diabetic Kidney Disease.

The role of glomerular morphometric features in pediatric podocytopathies - a single center study.

Sitagliptin and shock wave-supported peripheral blood derived endothelial progenitor cell therapy effectively preserves residual renal function in chronic kidney disease in rat-role of dipeptidyl peptidase 4 inhibition.

Tripterygium glycoside protects diabetic kidney disease mouse serum-induced podocyte injury by upregulating autophagy and downregulating β-arrestin-1.

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