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「"Tarlinton DM"[Author]」の検索結果

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Environmental sensing by mature B cells is controlled by the transcription factors PU.1 and SpiB.

Dynein light chain regulates adaptive and innate B cell development by distinctive genetic mechanisms.

IL4 and IL21 cooperate to induce the high Bcl6 protein level required for germinal center formation.

The life and death of immune cell types: The role of BCL-2 anti-apoptotic molecules.

The BTG2-PRMT1 module limits pre-B cell expansion by regulating the CDK4-Cyclin-D3 complex.

c-Myb Regulates the T-Bet-Dependent Differentiation Program in B Cells to Coordinate Antibody Responses.

Anti-apoptotic proteins BCL-2, MCL-1 and A1 summate collectively to maintain survival of immune cell populations both in vitro and in vivo.

MCL-1 is required throughout B-cell development and its loss sensitizes specific B-cell subsets to inhibition of BCL-2 or BCL-XL.

Innate Allorecognition Results in Rapid Accumulation of Monocyte-Derived Dendritic Cells.

Dynamic changes in Id3 and E-protein activity orchestrate germinal center and plasma cell development.

The Transcription Factor ASCIZ and Its Target DYNLL1 Are Essential for the Development and Expansion of MYC-Driven B Cell Lymphoma.

Regulation of germinal center responses, memory B cells and plasma cell formation-an update.

Glucocorticoid-induced leucine zipper (GILZ) inhibits B cell activation in systemic lupus erythematosus.

Targeting Antigen to Clec9A Primes Follicular Th Cell Memory Responses Capable of Robust Recall.

How many memories does it take to make an SLE flare?

c-Myb is required for plasma cell migration to bone marrow after immunization or infection.

Manipulation of B-cell responses with histone deacetylase inhibitors.

Transcriptional profiling of mouse B cell terminal differentiation defines a signature for antibody-secreting plasma cells.

Prosurvival Bcl-2 family members reveal a distinct apoptotic identity between conventional and plasmacytoid dendritic cells.

The generation of antibody-secreting plasma cells.

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