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「"Patel KP "[Author]」の検索結果

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RAS and TP53 can predict survival in adults with T-cell lymphoblastic leukemia treated with hyper-CVAD.

Lidocaine tripotassium phosphate complex laden microemulsion for prolonged local anaesthesia: In vitro and in vivo studies.

Central angiotensin II-Protein inhibitor of neuronal nitric oxide synthase (PIN) axis contribute to neurogenic hypertension.

Printed 3-Dimensional Computed Tomography Scanned Ankle Fractures as an Educational Instrument.

Comparison of therapy-related myelodysplastic syndrome with ring sideroblasts and de novo myelodysplastic syndrome with ring sideroblasts.

Central Glucagon-like Peptide-1 Receptor Signaling via Brainstem Catecholamine Neurons Counteracts Hypertension in Spontaneously Hypertensive Rats.

A Novel Disease Risk Model for Patients with AML Receiving Allogeneic Hematopoietic Cell Transplantation.

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Successful Lenalidomide Treatment in High Risk Myelodysplastic Syndrome with Germline DDX41 Mutation.

Does glucagon-like peptide-1 (GLP-1) induce diuresis and natriuresis by modulating afferent renal nerve activity?

Mixed myeloid chimerism and relapse of myelofibrosis after allogeneic stem cell transplantation.

System-Based Intervention Using Electronic Medical Records and Its Potential to Decrease the Overuse of Transthoracic Echocardiography.

Clinical implications of cytogenetic heterogeneity in Philadelphia chromosome positive (Ph++) adult B cell acute lymPh+oblastic leukemia following tyrosine kinase inhibitors and chemotherapy regimens.

Association of gene mutations with time-to-first treatment in 384 treatment-naive chronic lymphocytic leukaemia patients.

TOX transcriptionally and epigenetically programs CD8 T cell exhaustion.

Data on rearrangement-driven chromosomal aberrations in myeloid malignancies.

TP53 mutations are common in mantle cell lymphoma, including the indolent leukemic non-nodal variant.

Deciphering the complexities of MECOM rearrangement-driven chromosomal aberrations.

DDX41 Mutations in Myeloid Neoplasms are Associated with Male Gender, TP53 Mutations and High-Risk Disease.

Routine sequencing in CLL has prognostic implications and provides new insight into pathogenesis and targeted treatments.

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