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「"Loyau S "[Author]」の検索結果

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Critical role of neutrophil extracellular traps (NETs) in patients with Behcet's disease.

Cleavage of anti-PF4/Heparin IgG by a bacterial protease and potential benefit in heparin-induced thrombocytopenia.

Microfluidic Modeling of Thrombolysis.

A mutation of the human EPHB2 gene leads to a major platelet functional defect.

Carboxypeptidase U (CPU, CPB2, TAFIa) inhibition stimulates the fibrinolytic rate in different in vitro models.

Immobilized fibrinogen activates human platelets through GPVI.

Long-term management of leukocyte adhesion deficiency type III without hematopoietic stem cell transplantation.

Thrombus Neutrophil Extracellular Traps Content Impair tPA-Induced Thrombolysis in Acute Ischemic Stroke.

Amino-Fucoidan as a Vector for rtPA-Induced Fibrinolysis in Experimental Thrombotic Events.

Bioavailability and Cardiovascular Effects of Adrenaline Administered by Anapen Autoinjector in Healthy Volunteers.

Exacerbation of Thromboinflammation by Hyperglycemia Precipitates Cerebral Infarct Growth and Hemorrhagic Transformation.

Alteplase Reduces Downstream Microvascular Thrombosis and Improves the Benefit of Large Artery Recanalization in Stroke.

Inhibition of Glycoprotein VI Clustering by Collagen as a Mechanism of Inhibiting Collagen-Induced Platelet Responses: The Example of Losartan.

Single platelets seal neutrophil-induced vascular breaches via GPVI during immune complex-mediated inflammation in mice.

Platelet glycoprotein VI binds to polymerized fibrin and promotes thrombin generation.

Upholding the T cell immune-regulatory function of CD31 inhibits the formation of T/B immunological synapses in vitro and attenuates the development of experimental autoimmune arthritis in vivo.

Collagen Can Selectively Trigger a Platelet Secretory Phenotype via Glycoprotein VI.

High-density Lipoprotein-based Therapy Reduces the Hemorrhagic Complications Associated With Tissue Plasminogen Activator Treatment in Experimental Stroke.

Role of vegetation-associated protease activity in valve destruction in human infective endocarditis.

The future of GPVI as an antithrombotic target.

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