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マツタケ、絶滅危惧種に指定 (デイリースポーツ)

国際自然保護連合(IUCN)は9日、世界の絶滅危惧種をまとめたレッドリストの最新版を公表した。日本に関連する動植物では、新たに高級キノコのマツタケや沖縄県に生息...

  1. 第1回薬剤師の養成及び資質向上等に関する...
  2. 加藤厚生労働大臣 閣議後記者会見のお知ら...
  3. マツタケ、絶滅危惧種に 栽培困難、山の環...
  4. 囲碁 井山三冠 本因坊戦9連覇 七大タイ...

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「"Craigen WJ"[Author]」の検索結果

164件中 1件~20件表示    検索結果をPubMedで見る PubMedで見る

Response to Bai et al.

Deletion of VDAC1 Hinders Recovery of Mitochondrial and Renal Functions After Acute Kidney Injury.

Correction: Interpretation of mitochondrial tRNA variants.

Identification of a new aggressive axis driven by ciliogenesis and absence of VDAC1-ΔC in clear cell Renal Cell Carcinoma patients.

Correction: Interpretation of mitochondrial tRNA variants.

Corrigendum: 2-Pyrrolidinone and Succinimide as Clinical Screening Biomarkers for GABA-Transaminase Deficiency: Anti-seizure Medications Impact Accurate Diagnosis.

Interpretation of mitochondrial tRNA variants.

Aberrant Function of the C-Terminal Tail of HIST1H1E Accelerates Cellular Senescence and Causes Premature Aging.

Pathogenic Variants in Fucokinase Cause a Congenital Disorder of Glycosylation.

Unique aspects of sequence variant interpretation for inborn errors of metabolism (IEM): The ClinGen IEM Working Group and the Phenylalanine Hydroxylase Gene.

Development of Clinical Domain Working Groups for the Clinical Genome Resource (ClinGen): lessons learned and plans for the future.

MIRO-1 Determines Mitochondrial Shape Transition upon GPCR Activation and Ca Stress.

Correction: Sequence Homology at the Breakpoint and Clinical Phenotype of Mitochondrial DNA Deletion Syndromes.

Use of Exome Sequencing for Infants in Intensive Care Units: Ascertainment of Severe Single-Gene Disorders and Effect on Medical Management.

Novel Genetic Triggers and Genotype-Phenotype Correlations in Patients With Left Ventricular Noncompaction.

Mitochondrial Dysfunction Mediated by Poly(ADP-Ribose) Polymerase-1 Activation Contributes to Hippocampal Neuronal Damage Following Status Epilepticus.

Milder clinical and biochemical phenotypes associated with the c.482G>A (p.Arg161Gln) pathogenic variant in cobalamin C disease: Implications for management and screening.

YY1 Haploinsufficiency Causes an Intellectual Disability Syndrome Featuring Transcriptional and Chromatin Dysfunction.

De Novo Disruption of the Proteasome Regulatory Subunit PSMD12 Causes a Syndromic Neurodevelopmental Disorder.

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