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Anaerobic Glycolysis Maintains the Glomerular Filtration Barrier Independent of Mitochondrial Metabolism and Dynamics.

Early podocyte injury and elevated levels of urinary podocyte-derived extracellular vesicles in swine with metabolic syndrome: Role of podocyte mitochondria.

FHL2 mediates podocyte Rac1 activation and foot process effacement in hypertensive nephropathy.

Podocyte-Specific Sialylation-Deficient Mice Serve as a Model for Human FSGS.

[Dexamethasone Blocks Adriamycin-induced Podocytes'Mobility via Impacting Nephrin Expression].

Berberine Protects Glomerular Podocytes via Inhibiting Drp1-Mediated Mitochondrial Fission and Dysfunction.

miR-15b-5p ameliorated high glucose-induced podocyte injury through repressing apoptosis, oxidative stress, and inflammatory responses by targeting Sema3A.

Baicalein ameliorates pristane-induced lupus nephritis via activating Nrf2/HO-1 in myeloid-derived suppressor cells.

Meprin β metalloproteases associated with differential metabolite profiles in the plasma and urine of mice with type 1 diabetes and diabetic nephropathy.

Lipoprotein modulation of proteinuric renal injury.

Ghrelin modulates intracellular signalling pathways that are critical for podocyte survival.

Gut microbiome-derived phenyl sulfate contributes to albuminuria in diabetic kidney disease.

CD147/Basigin deficiency prevents the development of podocyte injury through FAK signaling.

Autophagy activity and expression pattern of autophagy-related markers in the podocytes of patients with lupus nephritis: association with pathological classification.

Corticosteroid treatment exacerbates nephrotic syndrome in a zebrafish model of magi2a knockout.

An unexpected role of steroid on podocytes: from zebrafish to human nephrotic syndrome?

Establishment of a Monoclonal Antibody PMab-231 for Tiger Podoplanin.

Hepatitis B virus X protein decreases nephrin expression and induces podocyte apoptosis via activating STAT3.

Blocking P2X7 receptor ameliorates oxidized LDL-mediated podocyte apoptosis.

C1q nephropathy in adults is a form of focal segmental glomerulosclerosis in terms of clinical characteristics.

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