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7547件中 181件~200件表示    検索結果をPubMedで見る PubMedで見る

Once-Daily Low-Dose Cyclosporine A Treatment with Angiotensin Blockade for Long-Term Remission of Nephropathy in Frasier Syndrome.

Integration of Genetic Testing and Pathology for the Diagnosis of Adults with FSGS.

Cadmium Induces Glomerular Endothelial Cell-Specific Expression of Complement Factor H via the -1635 AP-1 Binding Site.

Therapeutic Inhibition of VEGF Signaling and Associated Nephrotoxicities.

Altered VEGF Splicing Isoform Balance in Tumor Endothelium Involves Activation of Splicing Factors Srpk1 and Srsf1 by the Wilms' Tumor Suppressor Wt1.

Pharmacological Levels of Hydrogen Sulfide Inhibit Oxidative Cell Injury through Regulating the Redox State of Thioredoxin.

The relationship of anti-phospholipase A2 receptor antibody and C5a complement with disease activity and short-term outcome in idiopathic membranous nephropathy.

Manipulation of Nephron-Patterning Signals Enables Selective Induction of Podocytes from Human Pluripotent Stem Cells.

Producing Purer Podocytes.

Vitamin D protects podocytes from autoantibodies induced injury in lupus nephritis by reducing aberrant autophagy.

Complement Activation Contributes to the Pathophysiology of Shiga Toxin-Associated Hemolytic Uremic Syndrome.

[Regulatory Effect of Chinese Drugs for Stasis Removing and Collaterals Dredging on the Expres- sions of podocin and CD2AP in Podocyte Slit Diaphragm of Diabetic Nephropathy Rats].

Organoid single cell profiling identifies a transcriptional signature of glomerular disease.

Antigen-Specific IgG Subclasses in Primary and Malignancy-Associated Membranous Nephropathy.

Endothelium structure and function in kidney health and disease.

Serum phospholipase A2 receptor antibodies and immunoglobulin G subtypes in adult idiopathic membranous nephropathy: Clinical value assessment.

Aspirin attenuates podocyte injury in diabetic rats through overriding cyclooxygenase-2-mediated dysregulation of LDL receptor pathway.

Mesenchymal Stem Cell-Based Therapies against Podocyte Damage in Diabetic Nephropathy.

Ginseng Total Saponin Attenuates Podocyte Apoptosis Induced by Diabetic Conditions Through the Recovery of CD2-Associated Protein.

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