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Combining SGLT2 Inhibition With a Thiazolidinedione Additively Attenuate the Very Early Phase of Diabetic Nephropathy Progression in Type 2 Diabetes Mellitus.

A CRISP(e)R view on kidney organoids allows generation of an induced pluripotent stem cell-derived kidney model for drug discovery.

Podocyte NADPH Oxidase 5 Promotes Renal Inflammation Regulated by the Toll-like Receptor Pathway.

Nephron specific knockin of the PIKfyve binding deficient Vac14L156R mutant results in albuminuria and mesangial expansion.

Exploring the Clinical and Genetic Spectrum of Steroid Resistant Nephrotic Syndrome: The PodoNet Registry.

Crosstalk between transforming growth factor-β1 and endoplasmic reticulum stress regulates alpha-smooth muscle cell actin expression in podocytes.

Experimental Models to Study Podocyte Biology: Stock-Taking the Toolbox of Glomerular Research.

An eQTL Landscape of Kidney Tissue in Human Nephrotic Syndrome.

Amelioration of Diabetic Nephropathy Using a Retinoic Acid Receptor β2 Agonist.

In glomerular cells of puromycin aminonucleoside nephrosis rats both phosphorylated and total STAT3 levels increased during proteinuria.

APOL1 risk variants and kidney disease: what we know so far.

Expression of Cathepsin L and Its Intrinsic Inhibitors in Glomeruli of Rats With Puromycin Aminonucleoside Nephrosis.

Novel Microscopic Techniques for Podocyte Research.

Urine-derived cells: a promising diagnostic tool in Fabry disease patients.

RANK Deficiency Ameliorates Podocyte Injury by Suppressing Calcium/Calcineurin/ NFATc1 Signaling.

Emodin mitigates podocytes apoptosis induced by endoplasmic reticulum stress through the inhibition of the PERK pathway in diabetic nephropathy.

What Is the Glomerular Ultrafiltration Barrier?

Rapamycin Reduces Podocyte Apoptosis and is Involved in Autophagy and mTOR/ P70S6K/4EBP1 Signaling.

Activation of Cyclooxygenase-2 by ATF4 During Endoplasmic Reticulum Stress Regulates Kidney Podocyte Autophagy Induced by Lupus Nephritis.

BMSCs and miR-124a ameliorated diabetic nephropathy via inhibiting notch signalling pathway.

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