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Corticosteroid treatment exacerbates nephrotic syndrome in a zebrafish model of magi2a knockout.

An unexpected role of steroid on podocytes: from zebrafish to human nephrotic syndrome?

Establishment of a Monoclonal Antibody PMab-231 for Tiger Podoplanin.

Hepatitis B virus X protein decreases nephrin expression and induces podocyte apoptosis via activating STAT3.

Blocking P2X7 receptor ameliorates oxidized LDL-mediated podocyte apoptosis.

C1q nephropathy in adults is a form of focal segmental glomerulosclerosis in terms of clinical characteristics.

APOL1 renal risk variants promote cholesterol accumulation in tissues and cultured macrophages from APOL1 transgenic mice.

CRISPR-Cas9-Mediated Correction of the G189R-PAX2 Mutation in Induced Pluripotent Stem Cells from a Patient with Focal Segmental Glomerulosclerosis.

Establishment of a monoclonal antibody PMab-225 against alpaca podoplanin for immunohistochemical analyses.

Salidroside ameliorates Adriamycin nephropathy in mice by inhibiting β-catenin activity.

Zhen-wu-tang ameliorates membranous nephropathy rats through inhibiting NF-κB pathway and NLRP3 inflammasome.

Enzyme Replacement Therapy Clears Gb3 Deposits from a Podocyte Cell Culture Model of Fabry Disease but Fails to Restore Altered Cellular Signaling.

Low-dose agalsidase beta treatment in male pediatric patients with Fabry disease: A 5-year randomized controlled trial.

Interleukin-17A blockade reduces albuminuria and kidney injury in an accelerated model of diabetic nephropathy.

Geniposide alleviates lipopolysaccharide-caused apoptosis of murine kidney podocytes by activating Ras/Raf/MEK/ERK-mediated cell autophagy.

Urinary Nephrin as a Biomarker of Glomerular Maturation and Injury Is Associated with Acute Kidney Injury and Mortality in Critically Ill Neonates.

Local miscommunications between glomerular cells as potential therapeutic targets for crescentic glomerulonephritides.

Thrombospondin Type 1 Domain-Containing 7A (THSD7A) Localizes to the Slit Diaphragm and Stabilizes Membrane Dynamics of Fully Differentiated Podocytes.

The role of vasodilator-stimulated phosphoprotein in podocyte functioning.

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