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The Role of Podocytes and Podocyte-Associated Biomarkers in Diagnosis and Treatment of Diabetic Kidney Disease.

Hypertension Induces Glomerulosclerosis in Phospholipase C Epsilon 1 Deficiency.

Loss of Roundabout Guidance Receptor 2 (Robo2) in Podocytes Protects Adult Mice from Glomerular Injury by Maintaining Podocyte Foot Process Structure.

Role of the renin-angiotensin system in kidney development and programming of adult blood pressure.

Glomerular endothelial derived vesicles mediate podocyte dysfunction: A potential role for miRNA.

Overexpression of Myo1e promotes albumin endocytosis by mouse glomerular podocytes mediated by Dynamin.

Genetic Disorders of the Glomerular Filtration Barrier.

Urinary ERdj3 and mesencephalic astrocyte-derived neutrophic factor identify endoplasmic reticulum stress in glomerular disease.

Gut microbiota dysbiosis-induced activation of the intrarenal renin-angiotensin system is involved in kidney injuries in rat diabetic nephropathy.

ECM Characterization Reveals a Massive Activation of Acute Phase Response during FSGS.

Podocytopathy and Nephrotic Syndrome in Mice with Podocyte-Specific Deletion of the Asah1 Gene: Role of Ceramide Accumulation in Glomeruli.

The benzo[b]fluoranthene in the atmospheric fine particulate matter induces mouse glomerular podocytes injury via inhibition of autophagy.

Podocyte-specific deletion of tubular sclerosis complex 2 promotes focal segmental glomerulosclerosis and progressive renal failure.

Clinical course and pathological findings of two late-onset Fabry hemizygous patients including mulberry cell counts after enzyme replacement therapy.

ARHGEF7 (-PIX) Is Required for the Maintenance of Podocyte Architecture and Glomerular Function.

APOL1 C-Terminal Variants May Trigger Kidney Disease through Interference with APOL3 Control of Actomyosin.

Advances of the experimental models of idiopathic membranous nephropathy (Review).

Curcumin Inhibited Podocyte Cell Apoptosis and Accelerated Cell Autophagy in Diabetic Nephropathy via Regulating Beclin1/UVRAG/Bcl2.

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