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Calcium-Sensing Receptor Stimulation in Cultured Glomerular Podocytes Induces TRPC6-Dependent Calcium Entry and RhoA Activation.

An inducible mouse model of podocin-mutation-related nephrotic syndrome.

Synaptopodin is upregulated by IL-13 in eosinophilic esophagitis and regulates esophageal epithelial cell motility and barrier integrity.

High content screening assay based discovery of paullones as novel podocyte protective agents.

Is there a pathogenic association between Fabry's disease and IgA nephropathy?

Inducible ATF3-NFAT axis aggravates podocyte injury.

Mutations in INF2 may be associated with renal histology other than focal segmental glomerulosclerosis.

Mature induced-pluripotent-stem-cell-derived human podocytes reconstitute kidney glomerular-capillary-wall function on a chip.

PKA/CREB signaling prevents adriamycin-induced podocyte apoptosis via upregulation of mitochondrial respiratory chain complexes.

Clinicopathologic characteristics of light chain proximal tubulopathy with light chain inclusions involving multiple renal cell types
.

Renal outcome in IgA nephropathy according to Oxford classification and ultrastructural analysis in a Brazilian center
.

Effect of echinacoside on kidney fibrosis by inhibition of TGF-β1/Smads signaling pathway in the db/db mice model of diabetic nephropathy.

Metformin overcomes high glucose-induced insulin resistance of podocytes by pleiotropic effects on SIRT1 and AMPK.

Graft Growth and Podocyte Dedifferentiation in Donor-Recipient Size Mismatch Kidney Transplants.

Interaction of CD80 with Neph1: a potential mechanism of podocyte injury.

Deriving and understanding the risk of post-transplant recurrence of nephrotic syndrome in the light of current molecular and genetic advances.

Sustained activation of guanylate cyclase-A with TDT, a natriuretic peptide derivative, exhibits cardiorenal protection in Dahl salt-sensitive hypertensive rats.

ShcA Adaptor Protein Promotes Nephrin Endocytosis and Is Upregulated in Proteinuric Nephropathies.

MYH9 E1841K Mutation Augments Proteinuria and Podocyte Injury and Migration.

The diabetes pandemic suggests unmet needs for 'CKD with diabetes' in addition to 'diabetic nephropathy'-implications for pre-clinical research and drug testing.

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