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「"van Nimwegen M "[Author]」の検索結果

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Recommendations for the Organization of Multidisciplinary Clinical Care Teams in Parkinson's Disease.

Notch signaling licenses allergic airway inflammation by promoting Th2 cell lymph node egress.

T cell receptor repertoire characteristics both before and following immunotherapy correlate with clinical response in mesothelioma.

Type II conventional DCs of asthmatic patients with frequent exacerbations have an altered phenotype and frequency.

Enhanced Bruton's tyrosine kinase in B-cells and autoreactive IgA in patients with idiopathic pulmonary fibrosis.

Genomewide association study of Parkinson's disease clinical biomarkers in 12 longitudinal patients' cohorts.

Genetic risk of Parkinson disease and progression:: An analysis of 13 longitudinal cohorts.

Lower extremity muscle pathology in myotonic dystrophy type 1 assessed by quantitative MRI.

Increased surface expression of NOTCH on memory T cells in peripheral blood from asthma patients.

T cells and ILC2s are major effector cells in influenza-induced exacerbation of allergic airway inflammation in mice.

Fibrocytes are increased in lung and peripheral blood of patients with idiopathic pulmonary fibrosis.

Increased T helper 17.1 cells in sarcoidosis mediastinal lymph nodes.

A pathophysiological role of PDE3 in allergic airway inflammation.

The Notch Pathway Inhibitor SAHM1 Abrogates the Hallmarks of Allergic Asthma.

TNFAIP3 levels in lung dendritic cells instruct Th2 or Th17 cell differentiation in eosinophilic or neutrophilic asthma.

Depletion of tumor-associated macrophages with a CSF-1R kinase inhibitor enhances antitumor immunity and survival induced by DC immunotherapy.

Notch signaling in T cells is essential for allergic airway inflammation, but expression of Notch ligands Jagged1 and Jagged2 on dendritic cells is dispensable.

Physical Therapy and Occupational Therapy in Parkinson's Disease.

Impaired survival of regulatory T cells in pulmonary sarcoidosis.

Decreased Cytotoxic T-Lymphocyte Antigen 4 Expression on Regulatory T Cells and Th17 Cells in Sarcoidosis: Double Trouble?

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