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運動は中高年から始めても遅くない (日経BP)

運動は総死亡、心血管疾患、癌のリスク低下に関係することが示されている。しかし、多くの研究では、ベースラインの運動量とアウトカムの関係を調べており、その後の生活習...

  1. 医学部受験「バブル」はひとまず終息しまし...
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  3. 転移性前立腺癌の治療はより早期に新規AR...
  4. しばしば誤嚥性肺炎の原因となる意外なあの...

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「"Nutt SL "[Author]」の検索結果

203件中 1件~20件表示    検索結果をPubMedで見る PubMedで見る

New players in the gene regulatory network controlling late B cell differentiation.

Polycomb repressive complex 2 is a critical mediator of allergic inflammation.

Transcription factors IRF8 and PU.1 are required for follicular B cell development and BCL6-driven germinal center responses.

A Regulatory Circuit Controlling the Dynamics of NFκB cRel Transitions B Cells from Proliferation to Plasma Cell Differentiation.

PU.1 controls fibroblast polarization and tissue fibrosis.

The cis-Regulatory Atlas of the Mouse Immune System.

Viral replicative capacity, antigen availability via hematogenous spread and high T:T ratios drive induction of potent neutralizing antibody responses.

Transcription Factor PU.1 Promotes Conventional Dendritic Cell Identity and Function via Induction of Transcriptional Regulator DC-SCRIPT.

CCR2 enhances CD25 expression by FoxP3 regulatory T cells and regulates their abundance independently of chemotaxis and CCR2 myeloid cells.

Hippo Pathway Kinase Mst1 Is Required for Long-Lived Humoral Immunity.

Transcription-factor-mediated supervision of global genome architecture maintains B cell identity.

Plasma cells: the programming of an antibody-secreting machine.

Cochaperone Mzb1 is a key effector of Blimp1 in plasma cell differentiation and β1-integrin function.

IMiDs through loss of Ikaros and Aiolos primes myeloma cells for daratumumab mediated killing by upregulation of CD38.

Mining the Plasma Cell Transcriptome for Novel Cell Surface Proteins.

PU.1 Is Required for the Developmental Progression of Multipotent Progenitors to Common Lymphoid Progenitors.

Transcription Factor Theft-PU.1 Caught Red-Handed.

Editorial overview: Lymphocyte development and activation.

LMP2 immunoproteasome promotes lymphocyte survival by degrading apoptotic BH3-only proteins.

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