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「"Helin K "[Author]」の検索結果

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Correction for Danovi et al., "Amplification of (or ) Directly Contributes to Tumor Formation by Inhibiting p53 Tumor Suppressor Activity".

Molecular Mechanisms Directing PRC2 Recruitment and H3K27 Methylation.

The chromatin binding protein Phf6 restricts the self-renewal of hematopoietic stem cells.

TET2 binding to enhancers facilitates transcription factor recruitment in hematopoietic cells.

PLZF targets developmental enhancers for activation during osteogenic differentiation of human mesenchymal stem cells.

HER2-positive breast-cancer cell lines are sensitive to KDM5 inhibition: definition of a gene-expression model for the selection of sensitive cases.

Aggressiveness of non-EMT breast cancer cells relies on FBXO11 activity.

Secondary hypertension in patients with saccular intracranial aneurysm disease: A population based study.

The histone demethylase Jarid1b mediates angiotensin II-induced endothelial dysfunction by controlling the 3'UTR of soluble epoxide hydrolase.

The tumor suppressor CIC directly regulates MAPK pathway genes via histone deacetylation.

Epigenetic control of IL-23 expression in keratinocytes is important for chronic skin inflammation.

Accurate H3K27 methylation can be established de novo by SUZ12-directed PRC2.

Oncohistones: drivers of pediatric cancers.

Polycystic kidney disease among 4,436 intracranial aneurysm patients from a defined population.

Maternal expression of the JMJD2A/KDM4A histone demethylase is critical for pre-implantation development.

EPC1/TIP60-mediated histone acetylation facilitates spermiogenesis in mice.

Impact of Young Age on the Presentation of Saccular Intracranial Aneurysms: Population-Based Analysis of 4082 Patients.

Neurofibromatosis type 1 is not associated with subarachnoid haemorrhage.

EZH2 is a potential therapeutic target for H3K27M-mutant pediatric gliomas.

Jarid2 binds mono-ubiquitylated H2A lysine 119 to mediate crosstalk between Polycomb complexes PRC1 and PRC2.

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