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Way out/way in: How the relationship between WRN and CDK1 may change the fate of collapsed replication forks.

著者 Palermo V , Rinalducci S , Sanchez M , Grillini F , Franchitto A , Pichierri P
Mol Cell Oncol.2017 ; 4(1):e1268243.
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Replication-dependent double-strand breaks (DSBs) are the main source of genomic instability as their inaccurate repair stimulates chromosomal rearrangements. In a recent work, we uncover a novel regulatory circuit that involves the Werner's syndrome helicase and CDK1, and that is essential for repair pathway choice at replication-dependent DSBs.
PMID: 28197541 [PubMed - in process]
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