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Hypoxia augments anoctamin 1-encoded calcium-activated chloride current in cardiac vascular endothelial cells of neonatal mouse.

著者 Wu MM , Lou J , Song BL , Gong YF , Li YC , Yu CJ , Wang QS , Ma TX , Ma K , Hartzell HC , Duan DD , Zhao D , Zhang ZR
Br J Pharmacol.2014 Apr 24 ; ():.
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The molecular identity of calcium-activated chloride channels (CaCCs) in vascular endothelial cells remains unknown. This study aims to identify whether anoctamin 1 (ANO1, also known as TMEM16A) functions as CaCC and whether hypoxia alters biophysical properties of ANO1 in mouse cardiac vascular endothelial cells (CVECs).
PMID: 24758567 [PubMed - as supplied by publisher]
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